2.7.1.30: glycerol kinase
This is an abbreviated version!
For detailed information about glycerol kinase, go to the full flat file.
Word Map on EC 2.7.1.30
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2.7.1.30
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glycerol-3-phosphate
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triglyceride
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adrenal
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hypoplasia
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dystrophy
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muscular
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adipose
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3-phosphate
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duchenne
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lipase
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phosphoenolpyruvate
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hexokinase
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x-linked
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dihydroxyacetone
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contiguous
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adipocytes
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triacylglycerols
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carboxykinase
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congenita
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gluconeogenesis
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glycerophosphate
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lipolysis
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hpr
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iiaglc
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1,6-bisphosphate
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hypogonadism
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co-immobilized
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phosphocarrier
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glyceroneogenesis
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hypogonadotropic
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glycosomes
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dissimilation
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triolein
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1,3-propanediol
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glucose-specific
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aquaglyceroporins
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d-glyceraldehyde
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phosphoenolpyruvate:sugar
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dhap
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phosphoenolpyruvate-dependent
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sn-glycerol-3-phosphate
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sn-glycerol
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drug development
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diagnostics
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synthesis
- 2.7.1.30
- glycerol-3-phosphate
- triglyceride
- adrenal
- hypoplasia
- dystrophy
- muscular
- adipose
- 3-phosphate
-
duchenne
- lipase
- phosphoenolpyruvate
- hexokinase
-
x-linked
- dihydroxyacetone
-
contiguous
- adipocytes
- triacylglycerols
-
carboxykinase
- congenita
-
gluconeogenesis
- glycerophosphate
-
lipolysis
- hpr
- iiaglc
- 1,6-bisphosphate
- hypogonadism
-
co-immobilized
-
phosphocarrier
-
glyceroneogenesis
-
hypogonadotropic
- glycosomes
-
dissimilation
- triolein
- 1,3-propanediol
-
glucose-specific
-
aquaglyceroporins
- d-glyceraldehyde
-
phosphoenolpyruvate:sugar
- dhap
-
phosphoenolpyruvate-dependent
- sn-glycerol-3-phosphate
- sn-glycerol
- drug development
- diagnostics
- synthesis
Reaction
Synonyms
AFUB_068560, ASTP
ECTree
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diagnostics
drug development
glycerol kinase 5 (GK5) is a potential therapeutic target for treatment of NSCLC with resistance to EGFR tyrosine kinase inhibitors
synthesis
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methodology of dosage of glycerol kinase by response surface modeling of the enzymatic reaction. This low cost method for glycerol kinase dosage in a sequence of reactions is of great importance for many industries, like food, sugar and alcohol. Response surface modeling shows to be an adequate approach for modeling the reaction and optimization of reaction conditions to maximize glycerol kinase activity
frameshift mutations in a hypervariable homopolymeric region of the glpK gene are a specific marker of multidrug resistance in clinical Mycobacterium tuberculosis isolates, and these loss-of-function alleles are also enriched in extensively drug-resistant clones. Drug examples are isoniazid (INH), rifampin (RIF), pyrazinamide (PZA), and ethambutol (EMB)
diagnostics
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frameshift mutations in a hypervariable homopolymeric region of the glpK gene are a specific marker of multidrug resistance in clinical Mycobacterium tuberculosis isolates, and these loss-of-function alleles are also enriched in extensively drug-resistant clones. Drug examples are isoniazid (INH), rifampin (RIF), pyrazinamide (PZA), and ethambutol (EMB)
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