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Literature summary for 1.6.3.1 extracted from

  • Wakisaka, Y.; Miller, J.D.; Chu, Y.; Baumbach, G.L.; Wilson, S.; Faraci, F.M.; Sigmund, C.D.; Heistad, D.D.
    Oxidative stress through activation of NAD(P)H oxidase in hypertensive mice with spontaneous intracranial hemorrhage (2008), J. Cereb. Blood Flow Metab., 28, 1175-1185.
    View publication on PubMedView publication on EuropePMC

Application

Application Comment Organism
medicine experimental model of spontaneous intracranial hemorrhage in transgenic mice expressing human renin and human angiotensinogen treated with high-salt diet and Nomega-nitro-L-arginine methyl ester. In these mice, NAD(P)H oxidase activity is significantly increased. Increased enzyme activity preceeds signs of spontaneous intracranial hemorrhage and increases further whith development of intracranial hemorrhage Mus musculus

Organism

Organism UniProt Comment Textmining
Mus musculus
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hypertensive mice with spontaneous intracranial hemorrhage
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