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BRENDA support chlorophyll(ide) b reductase

This is an abbreviated version!
For detailed information about chlorophyll(ide) b reductase, go to the full flat file.

Word Map on EC


71-hydroxychlorophyllide a
chlorophyllide b


BoNYC1, CBR, Chl b reductase, chlorophyll b reductase, chlorophyll(ide) b reductase, LpNYC1, More, NOL, NON-YELLOW COLORING 1, NON-YELLOW COLORING1, NYC-like, NYC1, NYC1-LIKE


     1 Oxidoreductases
         1.1 Acting on the CH-OH group of donors
             1.1.1 With NAD+ or NADP+ as acceptor
       chlorophyll(ide) b reductase


Expression on EC - chlorophyll(ide) b reductase

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Chlorophyll loss in fruit peel is greatly accelerated by postharvest ethylene fumigation, which greatly increases the expression of chlorophyll b reductase NYC and chlorophyllase Chlase. The increase in Chlase and NYC transcript abundance is only related to accelerated chlorophyll degradation in ethylene-induced degreening
cytokinins and 1-MCP inhibit developmental increase in BoNYC1 expression. Samples treated with heat show a significant lower relative expression of BoNYC1 in comparison to controls after 72 h but not after 120 h of storage
ethylene, via 2-chloroethylphosphonic acid (ethephon, an ethylene-releasing agent), enhances developmental increase in BoNYC1 expression. Samples treated with visible light show a higher increment of BoNYC1 expression in relation to controls after 72 h but the expression is similar to those of controls after 120 h. Expressions of NYC1 and its homologue, NOL, increase markedly under intense light conditions
expression of LpNYC1 is highly inducible by abscisic acid (ABA) and ethephon (ethylene releasing reagent). Transcription factors LpABI5, LpABF3, and LpEIN3 directly activate the expression of LpNYC1 by binding to its promoter
expression of LpNYC1 is suppressed by treatment with AVG (ethylene biosynthesis inhibitor)
NYC1 expression is repressed in the abscisic acid-insensitive mutants during embryogenesis