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Literature summary for 1.6.5.2 extracted from

  • Merker, M.P.; Audi, S.H.; Bongard, R.D.; Lindemer, B.J.; Krenz, G.S.
    Influence of pulmonary arterial endothelial cells on quinone redox status: effect of hyperoxia-induced NAD(P)H:quinone oxidoreductase 1 (2006), Am. J. Physiol., 290, L607-619.
    View publication on PubMed

Organism

Organism UniProt Comment Textmining
Bos taurus
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Source Tissue

Source Tissue Comment Organism Textmining
artery pulmonary arterial endothelial cell. Increase in enzyme protein and activity upon hyperoxia, i.e. exposure to 95% O2 for 48 h Bos taurus
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endothelium pulmonary arterial endothelial cell. Increase in enzyme protein and activity upon hyperoxia, i.e. exposure to 95% O2 for 48 h Bos taurus
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Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
duroquinone + NADPH in both normoxic and hyperoxia-exposed cells, enzyme is the dominant duroquinone reductase Bos taurus durohydroquinone + NADP+
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