Literature summary for 5.4.2.2 extracted from

Csutora, P.; Karsai, A.; Nagy, T.; Vas, B.; Kovacs, G.L.; Rideg, O.; Bogner, P.; Miseta, A.
Lithium induces phosphoglucomutase activity in various tissues of rats and in bipolar patients (2006), Int. J. Neuropsychopharmacol., 9, 613-619.

Search for more literature for 5.4.2.2

Application

Application	Comment	Organism
medicine	inhibition of phosphoglucomutase activity by chronic treatment with Li+ causes compensatory elevation of phosphoglucomutase activity in Li+-treated bipolar patients due to increased expression of PGM1 gene	Homo sapiens

Organism

Organism	UniProt	Comment	Textmining
Homo sapiens	-	Li+-treated bipolar patients	-
Rattus norvegicus	-	-	-

Source Tissue

Source Tissue	Comment	Organism	Textmining
brain	inhibition of phosphoglucomutase activity by chronic treatment with Li+ causes alterations of glucose-phosphate level with compensatory elevation of phosphoglucomutase activity	Rattus norvegicus	-
leukocyte	inhibition of phosphoglucomutase activity by chronic treatment with Li+ causes compensatory elevation of phosphoglucomutase activity in Li+-treated bipolar patients due to increased expression of PGM1 gene	Homo sapiens	-
liver	inhibition of phosphoglucomutase activity by chronic treatment with Li+ causes alterations of glucose-phosphate level with compensatory elevation of phosphoglucomutase activity	Rattus norvegicus	-
muscle	skeletal muscle and cardiac muscle, inhibition of phosphoglucomutase activity by chronic treatment with Li+ causes alterations of glucose-phosphate level with compensatory elevation of phosphoglucomutase activity	Rattus norvegicus	-

Use of this online version of BRENDA is free under the CC BY 4.0 license. See terms of use for full details.

Release 2023.1 (January 2023)