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EC Number General Information Commentary Reference
Display the reaction diagram Show all sequences 1.14.11.58physiological function a non-polar OlsC mutant does not synthesize hydroxylated ornithine lipid and complementation with a plasmid containing OlsC restores the wild-type phenotype. OlsC is not required for Brucella virulence in laboratory models 764749
Display the reaction diagram Show all sequences 1.14.11.58physiological function hydroxylase OlsC modifies two species of ornithine-containing lipids in vivo, presumably by hydroxylation. A mutant carrying a nonpolar deletion in OlsC is symbiotically defective, whereas overexpressed OlsC in the complemented strain provokes an acid-sensitive phenotype -, 745812
Display the reaction diagram Show all sequences 1.14.11.58physiological function ornithine lipid hydroxylase OlsC introduces a hydroxyl group at the 2 position of the secondary fatty acid of ornithine lipids. Mutants deficient in OlsC cause an increase in nodule number that is reverted by the deletion of hydroxylase OlsE. OlsC is important in conferring stress resistance -, 745798
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